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The phosphorylation on AMPK can activate SIRT-1 or PGC1α, inhibit NFκB, and indirectly suppress proinflammatory gene transcriptions blocking the synthesis of proinflammatory mediators (cytokines, chemokines, and adhesion molecules) [116,129,185]
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10.1038/s41598-021-97967-0 Summary Keywords heart failure, GLP-1, glucagon-like peptide-1, clinical trials, cardiovascular disease, cardiovascular outcome studies, diabetes, obesity Citation Kreiner FF, Hovingh GKK and von Scholten BJ (2022) The potential of glucagon-like peptide-1 receptor agonists in heart failure
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