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Insulin resistance causes stimulation of the lipolysis signal, such as cAMP-PKA, to activate phospho-hormone-sensitive lipase (p-HSL)

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Furthermore, there are little data assessing whether envisioned changes in levels of endogenous NPY [1–36] vs NPY [3–36] in circulation and/or tissues after DPP4 inhibition accounts for a component of the relevant biological effects observed after DPP4 inhibition in vivo