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Moreover, in animal models of neuroinflammation, GLP-1RAs have demonstrated antioxidant, antiapoptotic, and neurotrophic effects
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In isolated cardiomyocytes, GLP-1 receptor agonists activated PI3K/Akt, AMPK, and cAMP/PKA pathways while reducing NOX4-mediated oxidative stress, signalling cardioprotective effects by alleviating ER stress-induced apoptosis [51]
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